By Sebastian L. Johnston
bronchial asthma: An Atlas of research and administration is an illustrated textbook and reference for clinicians. Containing a hundred and forty four complete colour pages, it presents important visible info at the prognosis, indicators and prevention of bronchial asthma. wealthy with photographic pictures supported through ordinary descriptions and reasons of every challenge, the ebook is a pragmatic and available source that every one medical professionals treating this will locate helpful of their day-by-day perform.
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Additional info for Asthma: An Atlas of Investigation and Diagnosis
Sample text
Further refinements to the hygiene hypothesis stem from the suggestion that specific infections occurring at critical times in immune development are most important: • Mycobacterial infection. Some epidemiological studies have suggested an inverse relation between mycobacterial exposure and asthma/atopy. • Intestinal flora. The gut is a critical regulator of the immune response and adequate intestinal flora may be important in guarding against atopy. This is supported by the association between enteric infection (hepatitis A), family size and absence of atopy in Italian recruits12.
4 The common mould Aspergillus fumigatus. Common moulds are ubiquitous and their spores, produced by hyphae (see left side of picture), are allergenic. Outdoor fungi tend to sporulate in early autumn, or at other times depending on weather conditions. Fungi growing indoors in the damp may sporulate at any time. 5 Animal dander. org). fore, not to miss allergen triggers for asthma especially in children and adolescents. House dust mite allergy may be an important trigger in perennial asthma. Asthmatic symptoms related to animal dander are easily identified.
Other signalling pathways such as mitogen-activated or extracellular signal-regulated protein kinase (MEK)/extracellular signal-regulated protein kinase (ERK), activated by enterotoxin superantigens from Staphylococcus aureus, and p38 mitogen-activated protein (MAP) kinase have been shown to phosphorylate the corticosteroid receptor itself, altering its abilities to bind corticosteroid and translocate to the cell nucleus. It will be some time before this immense complexity of interactions is fully elucidated, but already these early findings suggest new strategies for clinical characterization of asthmatic people (smoking, dietary antioxidant intake, bacterial colonization of the airways) better to predict their responsiveness to corticosteroid therapy.