By James A. Michener, A. Grove Day
In an exhilarating number of nonfiction event tales, James A. Michener returns to the main astonishing position in the world: the islands that encouraged Tales of the South Pacific. Co-written with A. Grove Day, Rascals in Paradise offers pictures of ten scandalous women and men, a few notorious and a few neglected, together with Sam Comstock, a mutinous sailor whose delusions of grandeur grew to become a nightmare; Will Mariner, a golden-haired formative years who used his attraction to win over his captors; and William Bligh, the infamous HMS Bounty captain who won't were the monster background recollects him as. From lifelong buccaneers to lapsed noblemen, in Michener and Day’s able palms those rogues develop into the stuff of legend.
Praise for Rascals in Paradise
“The most sensible publication approximately these far-scattered islands that has seemed in many years . . . a portfolio of infrequent and ruthless personalities that's calculated to make the curliest hair stand directly on end.”
—The manhattan Times
“[Combines] learn and scholarship (A. Grove Day used to be a professor on the collage of Hawaii) with a present for spinning a yarn and depicting personality (Michener, journalist and novelist, wishes no introduction).”
—Kirkus Reviews
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Additional info for Rascals in Paradise
Example text
The relative positions of these start sites are compared with those of pea in Figure 10. Whether light changes the relative abundance of the transcripts detected by S1 nuclease analysis remains to be determined. , 1989a,b), onlya single transcription start site has been detected. , 1987a; Sharrock and Quail, 1989). The negative autoregulation that phytochrome exerts over the expression of its own phyA genes in monocots provides an excellent model system for investigating the molecular basis of phytochrome-controlled gene expression (Colbert, 1988).
In this case, only lesions with global effects on all members of the phy family, such as disrupted chromophore synthesis, would be expected to result in phytochrome-deficient phenotypes. One approach to overcoming this problem is to overexpress a heterologous phytochrome polypeptide in transgenic plants. In this way, it is hoped either that overexpression of the wild-type sequence itself will produce a phenotype, or that overexpression of mutagenized sequences will interfere with endogenous phytochrome function to produce a dominant-negative phenotype (Herskowitz, 1987).
2,6). In addition, the cluster of negatively-charged D and E residues is less extensive than for phyA, with the result that the local net negative charge for phyB and phyC is -3 versus -5 to -7 for the phyA sequences (Fig. 7). , (1988) have shown that E-404 here (E-354 their numbering) is preferentially cleaved by endoproteinase Glu-C in the Pfr form, suggesting that this segment of the polypeptide moves closer to the surface of the molecule upon photoconversion to the active form. It has been proposed that this movement might account for the observation that Pfr is more negatively charged than Pr.