By Luciano Martini
The Encyclopedia of Endocrine Diseases is an authoritative reference built by means of sixteen foreign specialists who recruited the world's most sensible scientists for contributions. This stellar reference paintings makes a speciality of approximately 500 endocrine ailments addressing every thing from acromeglay, diabetes, high blood pressure, osteoporosis, thyroid disorder, Von Hippel-Lindau ailment, unexplained weight reduction, to androgen comparable problems. This encyclopedia is a vital addition to any library assortment written to be obtainable for either the medical and non-clinical reader.
Associate Editors comprise: Eli Y. Adashi, John P. Bilezikian, George P. Chrousos, Junichi Fukata, Julianne Imperato-McGinley, Hiroo Imura, Antonio Liuzzi, Frank L. Moore, Daniel Porte, Jens F. Rehfeld, Ellis Samols, Gustav Schonfeld, Pierre C. Sizonenko, Wilmar M. Wiersinga.
Also on hand on-line through ScienceDirect - that includes huge searching, looking out, and inner cross-referencing among articles within the paintings, plus dynamic linking to magazine articles and summary databases, making navigation versatile and simple. for additional info, pricing techniques and availability stopover at www.info.sciencedirect.com.
* contains approximately 500 entries addressing all facets of endocrine diseases
* greater than 800 eminent researchers contributed from 25 varied international locations round the world
* greater than 900 figures and 450 tables displayed
* complete word list comprising 1500 definitions
* The Editor-in-Chief is former editor of the magazine Frontiers in Neuroendocrinology and has contributed to over forty books within the box of endocrinology
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In Cushing’s syndrome or congenital adrenal hyperplasia) can cause hyperplastic adrenal glands. In addition to HCG, ACTH, and its receptor ACTHR, local growth factors are important for steroidogenesis, growth, and development of the adrenal gland. Insulin-like growth factors 1 and 2 (IGF-1 and IGF-2), their receptors, and binding proteins are all expressed in the fetal adrenal. IGF-1 not only amplifies the effect of ACTH on the adrenal but also enhances steroid production of the adrenal gland by increasing the activities of 17a, 21-, and 11b-hydroxylase.
Koch, C. , and Chrousos, G. P. (2001). Is the diminuto/dwarf1 gene involved in physiologic adrenocortical size regulation and tumor formation? J. Clin. Endocrinol. Metab. 86(11), 5127–5129. , Goldstein, D. , Sabban, E. , and Kopin, I. J. (1995). Sympathoadrenal system in stress: Interaction with the hypothalamic–pituitary–adrenocortical system. Ann. N. Y. Acad. Sci. 771, 131–158. Margioris, A. N. and Chrousos, G. P. ) (2001). ” Humana Press, Totowa, NJ. , and Jaffe, R. B. (1997). Development and function of the primate fetal adrenal cortex.
The important mediators for these functions are steroid hormones from the fetal adrenal. Throughout gestation and postnatally, the fetal adrenal gland undergoes morphological and functional changes during its transformation to the adult adrenal gland. The progenitor cells of the adrenal cortex stem from a cell lineage that also leads to steroid-secreting cells of the gonads. By week 8 of gestation, the adrenal cortexforming progenitor cells build an inner cluster (the fetal zone) consisting of large eosinophilic steroidsecreting cells that express high levels of steroid 17a-hydroxylase (CYP17) and an outer zone (the definitive zone) consisting of cells that do not express Encyclopedia of Endocrine Diseases, Volume 1.