By G. V. Gill, Gareth Williams, John C. Pickup
Tough Diabetes is meant for diabetologists and endocrinologists who are looking to hold abreast of the advancements during this fast-changing global. It presents a distillation of medical knowledge that's desperately required within the absence of evidence-based instructions. This publication seeks to prevent rehashing the approved content material and dogma present in the superb textbooks of diabetes. It offers extra of a detailed insurance than is feasible in person overview articles. Its key target is to supply suggestions to clinicians concerning the such a lot topical and arguable features in their perform. presents counsel to clinicians concerning the such a lot topical and debatable facets in their practiceEach bankruptcy is written through knowledgeable within the box who may give a balanced evaluate of the topicAll info is as modern as attainable as a result of quickly creation of the e-book
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Diabetologia 1992; 35 (Suppl 1): A60. Bourn DM, Mann JI, McSkimming BJ, Waldron MA, Wischart JD. Impaired glucose tolerance and NIDDM. Does a lifestyle intervention program have an effect? Diabetes Care 1994; 17: 1311–19. Eriksson J, Lindstrom J, Valle T et al. Prevention of type II diabetes in subjects with impaired glucose tolerance: the Diabetes Prevention Study (DPS) in Finland: study design and 1-year interim report on the feasibility of the lifestyle intervention programme. Diabetologia 1999; 42: 793–801.
Impaired glucose toleranceais it relevant for cardiovascular disease? Diabetologia 1997; 40: S138–40. Haffner SM. The importance of hyperglycaemia in the nonfasting state to the development of cardiovascular disease. Endocrinol Rev 1998; 19: 583–92. Tominaga M, Eguchi H, Manaka H et al. Impaired glucose tolerance is a risk factor for cardiovascular disease but not impaired fasting glucose. Diabetes Care 1999; 22: 920– 4. Perry RC, Baron AD. Impaired glucose tolerance: why is it not a disease? [editorial] Diabetes Care 1999; 22: 883–5.
1). Prior to the development of IGT, a large cohort can be described as having compensated insulin resistance. This has been shown to be the case in several studies of normoglycaemic relatives of people with type 2 diabetes [17–20]. Progression Age Obesity Ethnicity Physical inactivity Normal glucose tolerance Weight reduction Physical activity Pharmacological agents Insulin resistance Impaired glucose tolerance Beta-cell dysfunction Type 2 diabetes Fig. 1 Factors influencing the progression from normal glucose tolerance to IGT and to diabetes.